论文部分内容阅读
目的调查2型糖尿病家系成员的牙周状况。方法共收集43个家系[167人,男性71人,女性96人,平均年龄为(49.2±12.2)岁],分别抽取静脉血,并选取每位受检者的6颗代表牙,记录每颗牙的6个位点的菌斑指数(plaque index,PLI)、牙龈出血指数(bleeding index,BI)、牙周探诊深度(probing depth,PD)、附着丧失(attachment loss,AL)并记录总牙数及失牙数。结果 43个家系中共有糖尿病患者101例,其中4例全口牙缺失,经牙周病问卷调查显示,均为牙齿松动自行脱落或拔除。97例糖尿病患者均患牙周炎,其中轻度牙周炎50例,中度牙周炎24例,重度牙周炎23例。48例非糖尿病者中牙龈炎5例,轻度牙周炎30例,中度牙周炎11例,重度牙周炎2例。糖尿病患者和非糖尿病者龈炎和轻、中、重度牙周炎患病率差异有统计学意义(x~2=17.96,P<0.005),糖尿病患者的 PD、AL 及缺失牙数均高于非糖尿病者,差异有统计学意义(P<0.05)。血糖控制不良的糖尿病患者 BI、AL 均显著高于血糖控制良好的糖尿病患者(P<0.05)。血糖控制良好的糖尿病患者 PLI、BI、PD 及 AL 略高于非糖尿病者,但差异无统计学意义。结论在糖尿病家系成员中糖尿病患者的牙周炎患病率明显高于非糖尿病者,牙周破坏程度亦明显重于非糖尿病者;血糖控制良好患者的牙周状况与非糖尿病者相似。
Objective To investigate the periodontal status of type 2 diabetic pedigree members. METHODS: A total of 43 pedigrees (167 males, 71 males and 96 females with an average age of (49.2 ± 12.2) years) were enrolled in this study. Venous blood was collected and six representative teeth of each subject were selected. The plaque index (PLI), bleeding index (BI), probing depth (PD), attachment loss (AL) and the total number of records Number of teeth and loss of teeth. Results A total of 101 patients with diabetes were found in 43 pedigrees, of which 4 cases had complete loss of mouth. The results of periodontal disease questionnaire survey showed that all the patients had loose or detached teeth. 97 cases of diabetic patients suffering from periodontitis, including mild periodontitis in 50 cases, moderate periodontitis in 24 cases, severe periodontitis in 23 cases. 48 cases of non-diabetic gingivitis in 5 cases, 30 cases of mild periodontitis, moderate periodontitis in 11 cases, severe periodontitis in 2 cases. The prevalence of gingivitis and mild, moderate and severe periodontitis in diabetic patients and non-diabetic patients were significantly different (x ~ 2 = 17.96, P <0.005). The mean number of PD, AL and missing teeth in diabetic patients were higher than those in diabetic patients Non-diabetic patients, the difference was statistically significant (P <0.05). Patients with poorly controlled glycemic control had significantly higher BI and AL than those with well-controlled glycemic control (P <0.05). Patients with well-controlled glycemia had a slightly higher PLI, BI, PD and AL than those without diabetes, but the difference was not statistically significant. Conclusion The prevalence of periodontitis in diabetics is significantly higher than that in nondiabetics in diabetics, and the duration of periodontal destruction is also significantly higher than that in nondiabetics. The periodontal status in diabetic patients with good glycemic control is similar to that in nondiabetics.