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实验以急性亚极量强度跑台运动为主要研究方法,研究急性亚极量强度运动,是否也能够诱发ISPS的产生,并导致机体免疫机能的变化。研究结论如下:(1)急性亚极量强度运动应激后24小时,受试者血清中sIL-2R值明显升高;(2)急性亚极量强度运动应激后24小时,ISPS水平升高,对正常小鼠ConA诱导的淋巴细胞增殖产生明显的抑制作用;(3)急性亚极量强度运动应激后24小时,受试者机体的免疫功能受到抑制。实验证明了在急性亚极量强度运动应激24小时后,与运动前相比,运动后24小时,sIL-2R表达明显升高,表明机体内T淋巴细胞处于过度激活状态。与运动前相比,运动后淋巴细胞的转化受到明显的抑制,表明这种亚极量强度的运动可以导致机体内ISPS水平升高;而ISPS和sIL-2R的增加可以使淋巴细胞的功能受到抑制,使运动后机体过度激活的免疫机能得以保护。研究结果为今后在运动实践中应用ISPS监测运动员免疫机能提供了进一步的理论基础。
Experiments with acute suboptimal intensity of treadmill exercise as the main research methods to study the acute submaximal intensity exercise can also induce the generation of ISPS and lead to changes in immune function. The conclusions are as follows: (1) Acute suboptimal intensity 24 hours after exercise stress, serum sIL-2R values were significantly increased subjects; (2) acute suboptimal intensity 24 hours after exercise stress, ISPS level rise High, on the normal mouse ConA induced lymphocyte proliferation had a significant inhibitory effect; (3) acute submaximal intensity 24 hours after exercise stress, the subject’s immune function was inhibited. Experiments show that after 24 hours of acute submaximal exercise stress, sIL-2R expression is significantly increased 24 hours after exercise compared with pre-exercise, indicating that T lymphocytes in the body are over-activated. Compared with pre-exercise, the conversion of post-exercise lymphocytes was significantly inhibited, indicating that this sub-maximal intensity of exercise can lead to increased levels of ISPS in the body; and ISPS and sIL-2R increase in lymphocyte function can be affected Inhibition, so that the body after over-activation of immune function to be protected. The results provide a further theoretical basis for the future application of ISPS to monitor the immune function of athletes in sports practice.