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A.J.Clark曾指出药物产生疗效是通过活体细胞中酶的作用,此思想指导下,Bueding等等学者深入地研究了锑剂和氨苯氧烷化合物对曼氏血吸虫糖代谢的作用机制,并进行综述报导(1957)(1955),本文补充其近年来的工作,和收集部分有关日本血吸虫生化研究加以申述。血吸虫寄生在人体和其他哺乳动物门静脉、肠系膜静脉、膀胱静脉和肝窦中。血吸虫的生活史中需要适当钉螺作为中间宿主,当颤毛幼虫脱离钉螺后,在水中与哺乳动物的皮肤接触而侵入体内。引起血吸虫病的寄生虫有三种:曼民血吸虫(Schistosoma mansoni);埃氏血吸虫(Schistosoma haematobium); 日本血吸虫(Schistosoma japonicum)。分别地分布在埃及及其他非洲地区、中美洲、南美洲、中东、中国、日本和太平洋上某些岛屿。
AJClark has pointed out that the drug produces efficacy through the action of enzymes in living cells. Under the guidance of this thought, Bueding et al. Have deeply studied the mechanism of action of antimony agents and amnoxane compounds on the glycometabolism of Schistosoma mansoni and summarized the It is reported (1957) (1955) that this paper supplements its work in recent years and collects some of the biochemical studies on Schistosoma japonicum for inclusion. Schistosome parasites in the human body and other mammalian portal vein, mesenteric vein, bladder veins and sinusoids. In the life history of schistosomiasis, snails need to be used as an intermediate host. When the larvae of the schistosome detached from the snails, they invaded the body in contact with the mammalian skin in water. There are three parasites that cause schistosomiasis: Schistosoma mansoni; Schistosoma haematobium; Schistosoma japonicum. Respectively distributed in Egypt and other African regions, Central America, South America, the Middle East, China, Japan and the Pacific islands.