论文部分内容阅读
研究青藏铁路车厢内供氧是否能减轻缺氧对列车员脑组织的损伤。采用自主开发的近红外组织氧无损监测仪检测了在低海拔(平均海拔0.5～2.0km)路段和车厢内供氧的高海拔(平均海拔3.0～5.0 km,车厢内氧浓度维持在23%～25%)路段的列车员脑组织氧饱和度(rSO2),同时用脉搏血氧仪无创检测列车员指端脉搏血氧饱和度(SpO2)。高海拔路段同低海拔路段相比,rSO2平均下降3.6%,SpO2平均下降3.7%。这表明:车厢内供氧缓解了高原缺氧,但因个别列车员脑组织rSO2仍显著下降,所以对青藏铁路列车员的脑保护仍待研究。
To study whether the oxygen supply inside the Qinghai-Tibet Railway can reduce the brain damage caused by hypoxia. Infrared oxygen sensor was used to measure the oxygen supply altitude (mean elevation: 3.0 ~ 5.0 km) and the oxygen concentration in the cabin maintained at 23% ~ 25%) sections of the conductor brain tissue oxygen saturation (rSO2), while pulse oximetry non-invasive detection of the conductor fingertip pulse oxygen saturation (SpO2). The rSO2 decreased 3.6% on average and SpO2 dropped 3.7% on average in the high altitude section compared to the low altitude section. This indicates that the oxygen supply in the cabin alleviates the plateau hypoxia, but the cerebral protection of the Qinghai-Tibet Railway conductor remains to be studied because the rSO2 in the brain of the individual conductor is still significantly decreased.