目的　探讨基底节性失语的临床特点及发病机制。方法　选择经CT扫描证实为单侧基底节区病变患者 6 0例 ,在发病 2周 ,1月及 2月分别进行失语症检查。同时 ,进行BEAM及CT图象标准化处理。结果　基底节性失语患者病灶多在基底节区偏外侧 ,偏前部及偏上部。病变偏外侧者脑电地形图异常率高 ,且此类患者多有较重的听理解障碍 ,而尾状核受损者失语中有明显的构音障碍及音韵障碍。结论　基底节性失语有其临床特点 ,导致失语的机制多与基底节病变直接或间接影响皮层语言区 (血流量减少、代谢低下 )有关 ,但尾状核似可作为言语的皮层下整合中枢 ,在部分患者的失语中起作用 Objective To investigate the clinical features and pathogenesis of basal ganglia aphasia. Methods Sixty patients diagnosed as unilateral basal ganglia lesions by CT scan were selected. Aphasia examination was performed at 2 weeks, 1 month and 2 months respectively. At the same time, BEAM and CT image standardization. Results Basal aphasia patients mostly in the basal ganglia lesions outside the partial frontal and partial upper. Extra-lateral lesions of the EEG topography abnormal rate, and such patients have more severe hearing disorders, and atelectasis in caudate nucleus significant dysarthria and phonological disorders. Conclusions Basal ganglia aphasia has its clinical features. The mechanisms that lead to aphasia mostly relate to the changes of basal ganglia, which directly or indirectly affect the cortical linguistic area (blood flow decrease and hypometabolism). However, the caudate nucleus may serve as the cortical subdivision of speech center, In some patients aphasia play a role