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作者首先追述了氯硫二苯胺从胃肠道外输入正常人或实验动物能够引起排尿增多的现象,但不会同时增加电解质(钠与钾等)排泄量的事实。这种现象已被认为是与抗利尿物质(素),在此同时,在尿中排泄减少的缘故。作者因为充血性心力衰竭病人已被认为有抗利尿物质的大量排泄,故欲研究氯硫二苯胺对充血性心力衰竭病人的利尿作用。乃选用了三组不同性质的病人。第一组:共7人。为无心、肾疾病的其他疾病患者,用作对照。第二组:共5人。系心力衰竭者。其中3人为高血压性心脏病,2人为主动脈闭锁不全患者。
The authors first recount the fact that the introduction of chlorothiadiphenylamine into the normal or experimental animals from the outside of the gastrointestinal tract can cause increased urination but without simultaneously increasing the excretion of electrolytes (sodium and potassium, etc.). This phenomenon has been considered with anti-diuretic substances (Su), at the same time, reduced urinary excretion. Since the congestive heart failure patients have been considered a large excretion of antidiuretic substances, so want to study the chlorothiadiophenic diuretics in patients with congestive heart failure. Is the selection of three different groups of patients. The first group: a total of 7 people. Inadvertently, other diseases of kidney disease patients, as a control. The second group: a total of 5 people. Department of heart failure. Three of them were hypertensive heart disease and two were patients with aortic atresia.