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抗利尿激素异常分泌综合症(SIADH)的特点为水潴留引起的低钠血症和与低血浆钠浓度不相适应的尿钠排泄。SIADH病人,水潴留能明显地增加钠的排泄,但在上述情况下调节尿钠排泄的机理尚未阐明。为确定体液因素是否在钠排泄中起一定的作用,本文作者对由于肺燕麦细胞癌引起的SIADH所产主的低钠血症患者血浆中心房利钠因子进行测定。患者男性,60岁,分别在基础状态及水负荷情况下测定。在适当限制饮水条件下,血清电解质水平为:血钠129mEq/1;血钾3.7mEq/1;
Antidiuretic hormone hypersecretion syndrome (SIADH) is characterized by hyponatremia due to water retention and natriuresis excretion incompatible with low plasma sodium concentrations. SIADH patients, water retention can significantly increase the excretion of sodium, but in these cases the mechanism of regulating urinary sodium excretion has not been elucidated. To determine whether humoral factors play a role in sodium excretion, the authors determined plasma concentrations of natriuretic factor in SIADH-induced hyponatremia patients due to pulmonary oat cell carcinoma. Male patient, 60 years old, were measured under basic conditions and water load, respectively. Under appropriate drinking water conditions, serum electrolytes were: sodium 129 mEq / s; potassium 3.7 mEq / 1;