重症肌无力(MyG)是一种神经肌肉异常以随意肌的无力及易疲劳为特点。本病的临床特征虽早已为人们所熟知,但直至最近四年始明确其根本性损害是由于自身免疫所致的神经-肌肉接头处有效乙酰胆碱受体的减少。 MyG is a neuromuscular abnormality characterized by weakness and fatigue of voluntary muscles. Although the clinical features of this disease have long been known, it was not until the last four years that its underlying cause was the loss of effective acetylcholine receptors at the neuromuscular junction due to autoimmunity.