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近年来炎性反应在经皮冠状动脉介入治疗术后再狭窄中的作用受到重视 ,已证实在血管紧张素Ⅱ (AngⅡ )等因子作用下 ,损伤动脉中白细胞介素 6 (IL 6 )等炎性细胞因子产生增加 ,刺激中膜血管平滑肌细胞 (VSMC)向内膜移行、增殖并产生大量细胞外基质 ,导致冠状动脉介入治疗术后再
In recent years, the role of inflammatory response in restenosis after percutaneous coronary intervention has received great attention. It has been demonstrated that inflammatory factors such as interleukin 6 (IL 6) in the injured arteries are attenuated by angiotensin Ⅱ (AngⅡ) and other factors Increased production of cytokines stimulates the migration of tunica media vascular smooth muscle cells (VSMCs) to the intima, proliferation and production of extracellular matrix, leading to post-coronary intervention