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近年来研究表明,由自由基引发生物膜的脂质过氧化损伤是一种新的致病因素,可能是多种毒物毒性作用的起点。有学者认为,S_iO_2粉尘可引发和启动肺泡巨噬细胞和成纤维细胞的脂质过氧化损伤,从而促进矽肺病变的形成和发展,这为矽肺发病机理的深入研究,进而寻求有效的预防和治疗尘肺途径开辟了新的思
In recent years, studies have shown that lipid peroxidation damage caused by free radical biofilms is a new virulence factor that may be the starting point of a variety of toxic effects. Some scholars believe that, S_iO_2 dust can trigger and start alveolar macrophages and fibroblasts lipid peroxidation damage, thereby promoting the formation and development of silicosis, which in-depth study of the pathogenesis of silicosis, thus seeking effective prevention and treatment Pneumoconiosis has opened up new thinking