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医学论坛网报道,迟发性运动障碍的病理学基础尚不明确,尽管其临床临床特征涉及基底神经节,但影像学研究尚未找到明确证据证明其与基底神经或其他脑部结构的体积改变有关。《英国精神病学杂志》(The British Journal of Psychiatry)7月网络版刊登的一篇研究报告显示,迟发性运动障碍的病理学发展过程本质并非仅是一种神经化学反应,其亦可改变大脑结构。研究为确定精神分裂症伴迟发性运动障碍患者与无迟发性运动障碍患者相比,是否存在部分脑区的灰质体积改变,采用基于体素的结构成像技术对受试者进行必要的影像学检查。共有81名慢性精神分裂症患者参与研究,其中32人有49人无
Medical Forum Network reported that the pathological basis of tardive dyskinesia is not yet clear, although its clinical features related to the basal ganglia, but imaging studies have yet to find clear evidence of its basal nerve or other changes in the volume of brain structure . A study published in the July online edition of The British Journal of Psychiatry shows that the pathological development of tardive dyskinesia is not just a neurochemical reaction in nature, it can also alter the brain structure. To determine if there is a change in gray matter volume in some brain regions in patients with schizophrenia and tardive dyskinesia compared with patients without tardive dyskinesia, a voxel-based structural imaging technique is used to perform the necessary imaging of the subject School inspection. A total of 81 patients with chronic schizophrenia participated in the study, of which 32 were 49 without