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采用监测射频消融术（ＲＦＣＡ）前后的心肌酶及ＨＯＬＴＥＲ检查，对４２例房室折返性心动过速患者ＲＦＣＡ后出现的新的心律失常进行了评价。经心腔内电生理检查明确附加旁道的位置，其中２８例位于左游离壁，１４例位于间隔部位。大头导管详细标测消融“靶点”后给予ＲＦＣＡ，次数为２－４６，每例累积时间９０－１８７０（３８４±２５２）ｓ．ＲＦＣＡ均成功阻断旁道的前传及逆传，ＲＦＣＡ后第４天行ＨＯＬＴＥＲ检查，发现６例出现短阵房速，２例阵发性交界性心动过速，２例短阵室速，１例短阵室上速（该例患者再次心腔内电生理检查证实Ｖ：Ａ为１：１，系慢旁道所致，经再次ＲＦＣＡ后，无ＳＶＴ发作）．术前后ＨＯＬＴＥＲ检查室性期前收缩发生率在左游离壁旁道消融后有明显增加且存在显著差异；间隔部位旁道消融后最高心率及平均心率有明显加快亦有统计学意义。本文４２例术前查心肌酶均在正常范围，术后ＣＰＫ峰值在５７—６６９（３１２．８±２１４．０）ＩＵ／Ｌ，其中１８例位于正常范围。就上述结果可能的机理进行了讨论。
42 patients with atrioventricular reentrant tachycardia were assessed for arrhythmia after RFCA by monitoring cardiac enzymes and HOLTER before and after radiofrequency catheter ablation (RFCA). Transthoracic electrophysiological examination clearly attached to the location of the bypass, of which 28 cases located in the left wall, 14 cases located in the septal site. The RFCA was administered to the cuff after “ablation” of the cuff catheter and the number of RFCA was 2-46, with a cumulative time of 90-1870 (384 ± 252) s for each case. RFCA successfully blocked the anterior communicating and retrograde bypass, and the first 4 days after RFCA underwent HOLTER examination. There were 6 cases of atrial tachycardia, 2 cases of paroxysmal junctional tachycardia, 2 cases of short-term ventricular tachycardia, 1 Cases of paroxysmal supraventricular tachycardia (again patients with intracardiac electrophysiological examination confirmed that V: A is 1: 1, Department of slow bypass caused by the re-RFCA, no SVT attack). HOLTER preoperative and postoperative ventricular premature ventricular contractions in the left bypass wall after ablation increased significantly and there are significant differences; interval after bypass ablation of the maximum heart rate and average heart rate was significantly faster also statistically significant. 42 cases of preoperative myocardial enzymes were in the normal range, postoperative CPK peak 57-669 (312.8 ± 214.0) IU / L, of which 18 were in the normal range. The possible mechanisms for the above results are discussed.