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本文对在新建铁路隧道施工中长期接触一氧化碳的45名工人进行了血红蛋白和碳氧血红蛋白调查,并选择了大桥线路工38名为对照组。结果,调查组血红蛋白含量115～185g/L,平均149g/L,高于160g/L者13例;而对照组为105～140g/L,平均123g/L,无一例达160g/L,两组比较有显著性差异(P<0.05)。调查组碳氧血红蛋白平均0.119,明显高于正常围范。表明长期接触一氧化碳能造成机体组织的慢性缺氧,引起机体的代偿反应,使血红蛋白合成增加,血中含量升高。
In this paper, 45 workers exposed to carbon monoxide in the long-term construction of new railway tunnels were investigated for hemoglobin and carboxyhemoglobin, and 38 bridge workers were selected as the control group. Results: In the investigation group, hemoglobin content was 115-185g / L, with an average of 149g / L, higher than 160g / L in 13 cases; while the control group was 105-140g / L with an average of 123g / L, There was significant difference (P <0.05). The survey group carboxyhemoglobin average 0.119, significantly higher than the normal range. That long-term exposure to carbon monoxide can cause chronic hypoxia in the body tissues, causing the body’s compensatory response, hemoglobin synthesis increased blood levels.