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报告３３例慢性乙肝患者血浆血管紧张素Ⅱ（ＡＴ－Ⅱ）和醛固酮测定的结果。慢性迁延性肝炎和慢性活动性肝炎稳定期血浆（ＡＴ－Ⅱ）与正常对照没有差别，醛固酮比正常值增加２倍以上，无腹水的肝硬化病人ＡＴ－Ⅱ正常，而醛固酮比正常对照高２倍以上。有腹水的肝硬化病人ＡＴ－Ⅱ、醛固酮均较对照增高５倍以上。２５例慢性乙肝患者于速尿激发后ＡＴ－Ⅱ水平显著增高。以上结果提示腹水形成与有效血容量降低及肾素、ＡＴ－Ⅱ和醛固酮系统（ＲＡＡＳ）兴奋有关，故肝硬化失代偿期应用强利尿剂要有限量，以免造成严重后果。
Report of 33 cases of chronic hepatitis B patients with plasma angiotensin Ⅱ (AT-Ⅱ) and aldosterone determination results. The chronic persistent hepatitis and chronic active hepatitis with stable phase plasma (AT-Ⅱ) no difference from the normal control, aldosterone increased more than 2 times the normal, ascites cirrhosis patients AT-Ⅱ normal, and aldosterone than the normal control 2 Times more. Patients with cirrhosis with ascites AT-Ⅱ, aldosterone were increased more than 5 times higher than the control. 25 cases of chronic hepatitis B patients with furosemide stimulation AT-Ⅱ levels were significantly higher. The above results suggest that the formation of ascites and effective blood volume decreased and renin, AT-Ⅱ and aldosterone system (RAAS) excited, so the application of strong diuretic cirrhosis decompensation should be limited, so as to avoid serious consequences.