论文部分内容阅读
为从卵巢水平探讨运动性闭经的机制，选择动情周期正常的纯系雌性大鼠为实验对象，以大负荷游泳训练建立运动性动情周期抑制的动物模型。通过卵巢对人绒毛膜促性腺激素（hcG）兴奋试验，测定运动性动情周期抑制时卵巢中孕酮的变化。结果表明，卵巢对外源性促性腺激素hcG仍有敏感的反应，促进孕酮（P）的释放增加，提示卵巢分泌P的功能未发生不可逆的改变。
In order to explore the mechanism of exercise-induced amenorrhea at the ovarian level, pure-cycle female rats with normal estrous cycle were selected as experimental subjects, and animal models of exercise-induced estrous cycle inhibition were established by heavy-load swimming training. Ovarian progesterone (hcG) by the ovarian stimulation test to determine the kinetic estrous cycle inhibition of ovarian progesterone changes. The results showed that ovarian response to exogenous gonadotropin hcG still sensitive and promote the release of progesterone (P) increased, suggesting that the secretion of P in ovarian function did not irreversibly change.