17只麻醉狗在急性缺氧时,阻断低氧血对颈动脉体化学感受器的作用,可使肺动脉升压反应明显增强,肺血管阻力明显增高,肺血流量明显减少。反证了该化学感受器可能协调缺氧引起的肺动脉升压反应,使其升高不超过生理范围,即有助于改善肺血流灌注,促进机体对缺氧的适应,又可能防止肺动脉高压的形成。 17 anesthetized dogs in acute hypoxia, blocking hypoxia blood on the carotid body chemoreceptors, pulmonary vasopressor response was significantly enhanced pulmonary vascular resistance was significantly increased pulmonary blood flow decreased significantly. It turns out that this chemotherapeutic receptor may coordinate the pulmonary arterial hypertension response induced by hypoxia and make it rise beyond the physiological range, that is to say, it helps to improve pulmonary perfusion, promote the body’s adaptation to hypoxia, and may prevent the formation of pulmonary hypertension .